5 edition of Receptor science in cardiology found in the catalog.
Includes bibliographical references and index.
|Statement||edited by Jacob I. Haft and Joel S. Karliner.|
|Contributions||Haft, Jacob I., 1937-, Karliner, Joel S., 1936-|
|LC Classifications||QP114.R43 R43 1984|
|The Physical Object|
|Pagination||x, 286 p. :|
|Number of Pages||286|
|LC Control Number||83082169|
Thus, signalling through GPCRs regulates the degree of peripheral arterial resistance, aspects of renal function, the rate and force of myocardial contraction, and cardiac hypertrophy. 1 GPCRs involved in normal cardiovascular function include those that respond to angiotensin II (AT 1 receptors), to endothelin-1 (ET 1B receptors), and to Cited by: Search the world's most comprehensive index of full-text books. My libraryMissing: cardiology.
For Health Science Students Pharmacology Teferra Abula, Srinivasa , Amare Mengistu, Solomomon Worku, Eshetu Legesse, Musie Aberra, Dawit University of Gondar In collaboration with the Ethiopia Public Health Training Initiative, The Carter Center, the Ethiopia Ministry of Health, and the Ethiopia Ministry of Education * Elsevier is a leading publisher of health science books and journals, helping to advance medicine by delivering superior education, reference information and decision support tools to doctors, nurses, health practitioners and students.
Recent Trends in Cardiovascular Risks. All chapters in this book are related to the advances in cardiovascular risks. Chapters are useful for clinicians, medical practitioners, researchers, teachers, and students. Chapters include flow charts, diagrams, and tables for easy comprehension of overall risks of cardiovascular disease. Author(s. Cardiac cells, like those of the other tissues, undergo regulation through membrane-bound proteins known as G protein-coupled receptors (GPCRs). β-adrenergic receptors (βARs) are key GPCRs expressed on cardiomyocytes and their role is crucial in cardiac physiology since they regulate inotropic and chronotropic responses of the sympathetic nervous system (SNS).Cited by:
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Receptor Science in Cardiology 1st Edition by Jacob I. Haft (Editor), Joel S. Karliner (Editor) ISBN Additional Physical Format: Online version: Receptor science in cardiology.
Mount Kisco, N.Y.: Futura Pub. Co., (OCoLC) Document Type. Cardiac Receptors: Medicine & Health Science Books @ Skip to main content. Try Prime EN Hello, Sign in Account & Lists Sign in Account & Lists Returns & Orders Try Prime Cart.
Books. Go Search Hello Select your address. Book chapter Full text access Beta3-Adrenergic Receptors in Cardiac and Vascular Tissues: Emerging Concepts and Therapeutic Perspectives Chantal Dessy, Jean-Luc Balligand. This book draws together and analyzes the current literature to provide readers with a state-of-the-science review as well as current techniques that support research in all aspects of the field of lysophospholipid signaling.
Lysophospholipid Receptors is divided into three sections. Andrew S. French, Päivi H. Torkkeli, in Cell Physiology Source Book (Fourth Edition), Sensory receptors are specialized cells, usually neurons, that detect and respond to physical and chemical stimuli.
Most are exquisitely sensitive to highly specific inputs, or sensory modalities, such as movement, odor chemicals or visible light photons.
Braunwald's Heart Disease: A Textbook of Cardiovascular Medicine, 11th Edition Trusted by generations of cardiologists for the latest, most reliable guidance in the field, Braunwald's Heart Disease, 11th Edition, remains your #1 source of information on rapidly changing clinical science, clinical and translational research, and evidence-based /5(31).
Discover the best Receptor science in cardiology book in Best Sellers. Find the top most popular items in Amazon Books Best Sellers. This note covers the following topics related to Renovascular Hypertension treatment and medication: ACE Inhibitors, Angiotensin Receptor Blockers (ARBs), Blockers, Beta 1 Selective, Beta Blockers, Nonselective Alpha Blockers, Antihypertensives, Calcium Channel Blockers, Diuretics, Vasodilators, Cardiovascular, Pharmacologic Therapy, Percutaneous Transluminal Angioplasty, Surgical.
COVID is caused by the severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2), which invades cells through the angiotensin-converting enzyme 2 (ACE2) receptor.
Among those with COVID, there is a higher prevalence of cardiovascular disease and >7% of patients suffer myocardial injury from the infection (22% of the critically ill). The activation of the angiotensin-converting enzyme (ACE2)/Ang ()/Mas receptor axis and its beneficial role on cardiac structural and electrical remodeling is discussed.
Moreover, new developments including the presence of angiotensin () Ang (), neprilysin inhibitors, and its relevance for heart failure as well as (pro)renin receptors and their possible role on cardiovascular disease are discussed.
Patients with such comorbidities are commonly treated with renin angiotensin system blockers, such as angiotensin-converting enzyme inhibitors (ACEIs) or angiotensin-receptor blockers (ARBs). However, the use of ACEIs/ARBs in patients with COVID or at risk of COVID infection is currently a.
Propranolol is the prototypic non-subtype-selective beta antagonist which has equal affinities at the beta-1 and beta-2 subtypes. Other nonselective beta adrenergic antagonists include timolol, pindolol (which is actually a weak partial agonist), and carvedilol, which is also an alpha-1 antagonist.
The glucocorticoid receptor is a member of the nuclear receptor family that controls many distinct gene networks, governing various aspects of development, metabolism, inflammation, and the stress response, as well as other key biological processes in the cardiovascular system.
Recently, research in both animal models and humans has begun to unravel the profound complexity of glucocorticoid Cited by: 3. There are four subtypes of β-ARs-β 1-AR, β 2-AR, β 3-AR and the β 4-AR ().The β 1-AR is found primarily in the heart and comprises 75–80% of the β-ARs found in the heart (Figure 2).The β 2-AR is expressed in the lungs, kidneys and blood vessels as well as the heart and comprises 20–25% of cardiac β 3-AR is found primarily in the adipose tissue, and minimally in the by: Frontiers Books allows users to browse, free of charge, through the pages of Frontiers e-books and e-magazines or download the entire pdf.
You may also purchase hardcopy issues as well as other academic literature. The American Heart Association explains the various medications for heart disease and cardiovascular conditions, such as Anticoagulants, Blood Thinners, Antiplatelet Agents, Angiotensin-Converting Enzyme Inhibitors, ACE Inhibitors, Angiotensin II Receptor Blockers, Angiotensin II Receptor Inhibitors, Beta Blockers, Calcium Channel Blockers, Diuretics, Vasodilators, Nitrates, Nitroglycerin.
Our portfolio provides a wide range of journals, monographs, major reference works, textbooks and book series covering topics across the field of cardiology. From cutting edge original investigations to basic science, Springer Nature’s cardiology titles feature authors and contributions from key researchers and leading institutions around the.
Tissue tropism of SARS-CoV is stipulated by a specific receptor-facilitated process; angiotensin converting enzyme 2 (ACE2) has been identified as the cellular receptor that binds directly to the viral S protein. 51 ACE2 is expressed in alveolar epithelial cells and in surface enterocytes of the small intestine, both of which are the primary target cells of SARS-CoV infection.
32 ACE2, which acts as a negative. The continuing story of 5-hydroxytryptamine receptors: a 5-HT 3 receptor modulates dopamine release from rat striatal slice Patrizio Blandina, Joseph Goldfarb, Jack Peter Green Pages. The V 1 receptor. The V 1 R gene is located on chromosome 12 and maps to region 12q .Functionally, the V 1 R activates G-proteins of the G q/11 family.
The α-subunits regulate the activity of the β-isoforms of phospholipase C .A variety of signaling pathways is associated with the V 1 R, and these pathways include activation of calcium influx, phospholipase A 2, phospholipase C, and Cited by: Braunwald's Heart Disease: A Textbook of Cardiovascular Medicine, 11th Edition Trusted by generations of cardiologists for the latest, most reliable guidance in the field, Braunwald's Heart Disease, 11th Edition, remains your #1 source of information on rapidly changing clinical science, clinical and translational research, and evidence-based by: Clinical Topics: Diabetes and Cardiometabolic Disease, Dyslipidemia, Heart Failure and Cardiomyopathies, Prevention, Lipid Metabolism, Novel Agents, Acute Heart Failure, Heart Failure and Cardiac Biomarkers, Hypertension Keywords: Angiotensin II, Angiotensin Receptor Antagonists, Angiotensin-Converting Enzyme Inhibitors, Blood Pressure, COVID, Coronavirus, Heart Failure.